I can see that the aim of my question is rather far from the article content. However, I am just a surgeon, and the question is quite important in both practical and scientific point of view. Having much references browsed, I can not find a reliable answer.
Besides, you found that not less than 5% of the liver tissues studied contain the exocrine pancreatic tissue (which to some extent resembles that of teleost fish liver) [Terada T, Nakanuma Y, Kakita A. / Pathologic observations of intrahepatic peribiliary glands in 1,000 consecutive autopsy livers: Hetero-
topic pancreas in the liver // Gastroenterology 1990; 98: 1333-1337]. So, if you do not mind, my second question goes forth:
Why does the pancreatic juice inside intrahepatic bile ducts remains harmful and does not cause the chronic cholangitis, or something of the kind?
Article What are ductal plate and ductal plate malformations of human livers?
Thank you very much!
But... how it is possible for the monolayer to transport some bile? And when does it start, this process, and how it can be checked for sure?
I thought that the principle of treatment for the biliary athresia is a liver transplantation. On another hand, I am just an 'adult' surgeon.
Sorry! I am afraid, I have been mistaking hepatic congenital fibrosis with biliary agenesia!
For biliary agenesia, Kasai's intervention is the first step shortly after birth, followed by transplantation if not efficent enough.
Thank you! So, what is your opinion about the ductal plate structures' capacity to deliver bile into the duodenum?
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