There are many growth factors in a wound.  They have different roles and temporal appearances.  You must think a lot more specifically about the question.  

For example, in the liver, following injury, the first to activate is HGF from its latent form.  24 hrs later TGF-beta begins to come into play.    For the proper repair, both will be needed at different times.  Skin is even more complicated because many of the GFs are delivered by the blood cells in addition to what is in matrix and exposed by the wound.

Interesting question. I am sure not answered yet but worth of investigation. I imagine of the dressing containing some human plasma or saliva (HGF is abundant there).   

I would have thought start with a totally inert dressing with no cytotoxic properties e.g. Melolin or paraffin gauze.

Is collagen appropriate for this task?

The best is a non interactive, atraumatic on removal,  silicone mesh. It keeps growth factors in place (eg if you have applied Platelet rich plasma); allows for exudate to seep through that prevent dilution of the growth factor application and on removal do not disturb any new growth that did occur. Can be left in place for 5-7 days. Do not mechanically cleanse the wound bed on re-opening/ re-dressing after growth factor application, leave it in a atraumatic state.

Besides silicone coating, Biobrane, Mepitel; Omiderm etc. which is or are appropriate?

I would use some hydro-selective dressings like Cutinova. It is supposed to maintain growth factors in the needed area.

While I find your comments interesting from a clinical perspective, I still think  you are not identifying several key points.  What kind of wound?  (burn verses cut)  If cut, how deep?  Shallow wounds are best left to the air to promote keritinization process.  Deeper wounds need coverage for a while until basel layer reforms.  Deep cuts will require many more growth factors over a longer periods with very different temporal expression/appearance patterns.  Largely the initial attractor is the clot which is a major binder and attractor of the infiltrating leukocytes.  The platlets that stick to and rupture at the clot, release lots of different GFs.  

Here is a good review article detailing the process in the skin.

http://physrev.physiology.org/content/83/3/835.long

Thank you.

Will be used on widely meshed (1:3; 1:4) partial-thickness skin grafts, beginning from the postoperative 1 day.

In fresh wounds with defect, like fingertip Amputation injuries, covering the defekt with a semipermeable Dressing like op site maintaines a favorable Environment for healing with Regeneration of the fingertip if at least part of the germinative Matrix of the fingernail is preserved. It seems that an activated WnT pathway expressed by nail stem cells and neurotrophic factors from transected small nerves are responsible for at least partial Regeneration of the tip if treated by the method first published by Mennen  and Wiese.

J Hand Surg Br. 1993 Aug;18(4):416-22.

Fingertip injuries management with semi-occlusive dressing.

Mennen U1, Wiese A.

Handchir Mikrochir Plast Chir. 1998 Jan;30(1):24-9.

[Treatment of fingertip defect injuries with a semi-occlusive dressing].

[Article in German]

Quell M1, Neubauer T, Wagner M.

Nature. 2013 Jul 11;499(7457):228-32. doi: 10.1038/nature12214. Epub 2013 Jun 12.

Wnt activation in nail epithelium couples nail growth to digit regeneration.

Takeo M1, Chou WC, Sun Q, Lee W, Rabbani P, Loomis C, Taketo MM, Ito M.

I hope it might be interesting to You

MQ

Article [Fingertip reconstruction with occlusive dressing: Clinical ...

Article [Treatment of fingertip amputation with semiocclusive dressing].

Conference Paper Experience with semi-occlusive dressing in fingertip injurie...