For example, mice are infected with acute LCMV virus. Normally, CD8 T expand and reach effector function at day 7 when virus was cleared. I am wondering what happen if mice are reinfected with virus at day 7 post primary infection ? Does the effector CD8 T cells clear virus more rapid or these effector CD8T cells start activation induced death or become anergy due to re-exposure to strong antigen? Thanks