Thanks for your contributions. The question is: cows fed linseed produce milk with higher C18:3n3 but with really small increases in its typical content of Oemga-3 (because cows scarcely convert C18:3n3 into either DHA or EPA). Then, this is not useful for improving human health as humans cannot utilize C18:3n3 as a useful source of Omega-3, as we need EPA and DHA and we cannot convert C18:3n3 into EPA and DHA.
Milk fat contributes between 10-15% to daily intake of EPA and that is significant. Humans can convert C18:3n-3 into EPA and other C20 and C22 omega-3 fatty acids by the omega-3 pathway. Mammals that are not able to do that are cats etc.
Did you attend the last EAAP meeting? Dr. Bauman (if I am not wrong) highlighted this almost lack of capability by both humans and cows to convert C18:3n3 into DHA and EPA. Maybe we are facing either different data or conclusions. I will search for the talk and share it here. Interesting discussion.