Emotional vulnerability and having difficulty on an etiological manifestation of symptomology deals with sensitivity where in looking at neurology the receptors in the neurons (network) are stimulated where the balance of regulating emotions falls on a psychological basis in observation of strong reactions and returning to, per say, balanced behavior being slow on an abnormal side of linguistic in imagery and the baseline after calibration is considered normal. When the sufferer is observed in attempting to balance their emotions, due to on a biological basis modulated (introverted/introverted) aspect of the environment aspect of the environment, this can signify non-comorbidity with other conflicts or disorder depending on diagnosis. The symptomology deals with mild, moderate, and severe with examples like, erratic self-destructive, impulsion and/or from above comorbidity of changing their behavior to compensate to their environment. This is a point of “dementia, delusion, hallucination” in diagnostics within the environment whether controlled or not (researchive) these unstable patterns lead to intense relationships that can manifest the possible or hypothetical comorbidity. (ex. Personality, obsessive compulsive, addiction…). The amygdala deals with the negative natural states within dimensional neurology. This leads to fear and anxiety that modulates neuro-net vigilantes. This is considered co-occurring disorders as well as etiology to psychopathy (Donegan, Sanslow, Blumberg et.al., 2003).or what can be neutral

The pathways when medicine is needed are nasal administered as well as through the blood… where the medicine can enter the brain using theory of olfactory to the base lateral side of the epithelium. Here traces are discovered in the cerebrospinal fluid (CSF). This aspect of administration of medicine is quicker on a relative basis but can also be administered slowly into the spinal fluid. This medicine gets through the blood brain barrier with the use of liposomes. These are agonists points or what can be neutrally called the bio membrane. The diversity of sufferer’s biology to focusing on modification and objective. Anxiolytic point of cognitive evolutionary advancement effects leads to optimization through oral administration through oral administration to achieve baseline imagery in the neuro synapse in discovering dimension for anxiety and its cognitive affects (Pipren, Watanaton, Sutthparing, et.al., 2008).

When looking at cranial modification, foundational brain size theory comes to mind. This leads to environment or health practices assessment, analysis on morphism, and foundational biology (embodiment). On a researchive basis, it leads to evolutionary advancement in medicine but in associative linguistic, the direction of “Supremacy” comes to mind when looking at brain size theory dominate points of species in life in the environment. With terrorism today and empirical findings of genetic engineering on an invitro and nanotechnology basis, we find terrorism and cell growth of acclaimed armies of insulation (Axis) and turn over points in employment and professional arenas in exchange pursuit (s) of freedom. When looking at why cranial modification and how it was done it can go back to 400 BC with Hippocrates Hutchins 1952) was the first discovery that described the practice (Philosophy) {direction best interest, (Ex. Galan Theory (Circulation…)}. The skull of a newborn soft during development and the first points in empirical research of cranial modification was using bandages to form the development of the heads of new forming generations (children). This was a point of lapse of information until 1800 when many researchers and authors where categorizing the recent types of cranial modification (Duncan, & Hofling, 2011).