Meningeal contribution to migraine pain: a magnetic resonance angiography study
Sabrina Khan Faisal Mohammad Amin Casper Emil Christensen Hashmat Ghanizada Samaira Younis Anne Christine Rye Olinger Patrick J H de Koning Henrik B W Larsson Messoud Ashina Brain, Volume 142, Issue 1, 1 January 2019, Pages 93–102,https://doi.org/10.1093/brain/awy300
Published: 26 December 2018
The devil in migraine research is not in the details, as opposed to the untrammeled accumulation of data under the guise and the premise that migraine with aura and migraine without aura are different entities like London and Sydney.
While migraine most frequently affects the ophthalmic division of the trigeminal nerve, with dural stimulation and middle meningeal artery involvement, such a neural activation is not possible (Gupta, 2009).
The same wine keeps coming back in different bottles, and more often, in similar bottles. And above all, asks for cheers in the explanatory video from the dumb and dumber.
Migraine researchers simply refuse to learn from the past, or from neuroanatomy, or from neuropharmacology, or from the complete absence of meaning of data in the absence of an overarching salient generalizable and comprehensive matrix, breaking down the migraine syndrome into further syndromic artificial fragments to give meaning to data (Gupta 2009, 2010).
Articles that are attached as files to such critiques are never read, because the researchers/investigators are complete and compleat, by and in themselves.
The long and dark night of migraine research / the dark side of cortical spreading depression with silent cortical events like the dark side of the web keeps expanding as an intergalactic immense black hole, as investigators keep on testing their key against different locks, blindfolded.
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