I suggest you read these articles, 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3249970/ 

and http://www.sciencedirect.com/science/article/pii/S1016319016000240

and https://www.ajol.info/index.php/ajb/article/download/129984/119542 

and https://www.hindawi.com/journals/cjidmm/2016/9041636/

I suggest you also read this article which may be helpful

http://jmm.microbiologyresearch.org/content/journal/jmm/10.1099/jmm.0.47144-0#tab2

The emergence of vancomycin-resistant S. aureus (VRSA), is due to acquisition from Enterococcus spp. of the vanA operon, carried by transposon Tn1546, resulting in high-level resistance.

Three classes of VRSA  have emerged that differ in vancomycin susceptibilities: vancomycin-intermediate S. aureus (VISA), heterogeneous vancomycin-intermediate S. aureus (hVISA), and high-level vancomycin-resistant S. aureus (VRSA).

To be full VRSA, S aureus should has VanA, VanB. VanC, VanD, etc...., otherwise any S. aureus has only one or two genes of Van should be considered as Partially VRSA (VISA).

Regards 

Thanks my colleagues

Thank you Prof. Gamal. I 've run over your C.V, Its interesting.

Regards