Cause various cardiovascular events such as angina, heart attack, stroke and peripheral artery disease. Atheroma root cause of a number of cardiovascular diseases "angina, myocardial infarction, high blood pressure", cerebrovascular disease} {stroke, peripheral artery disease.

To prevent the build up of atheroma: {quit smoking may be appropriate}

Atheroma is also known as atherosclerosis and 'hardening of the arteries'.

Patches of atheroma are like small fatty lumps that develop within the inside lining of blood vessels (arteries). Patches of atheroma are often called plaques of atheroma and can make an artery narrower, which can restrict and reduce the blood flow through the artery

Atheroma is the cause of various cardiovascular diseases such as angina, heart attack, stroke and peripheral vascular disease. Atheroma is the root cause of a number of cardiovascular diseases (angina, myocardial infarction, hypertension), cerebrovascular disease (stroke), peripheral arterial disease.

To prevent a build-up of atheroma: stopping smoking, reducing a high blood cholesterol level, treating high blood pressure, eating a healthy diet, keeping your weight in check and doing some regular physical activity

One of the secrets is to have a healthy life

Why do arteries develop atheromas? Well, nature have some tools in order to impose the going of the old generation and the coming and the promotion of the new one! Atheroma is one of them. 

Cause various cardiovascular events such as angina, heart attack, stroke and peripheral artery disease. Atheroma root cause of a number of cardiovascular diseases "angina, myocardial infarction, high blood pressure", cerebrovascular disease} {stroke, peripheral artery disease.

To prevent the build up of atheroma: {quit smoking may be appropriate}

Atheroma - a fatty deposit within the inside lining of arteries- is the cause of most cardiovascular diseases. There are actions that can be done to reduce the risk of forming atheroma. These include :choosing healthy foods & minimizing saturated fats, a low sugar & salt intake, continuous physical activity, keeping the body's weight down,and giving up alcohol & smoking.

Dear Vilemar,

Atheroma is cause of a number of cardiovascular diseases. I think that in my country we are infomed about this.

I think that stopping smoking , reducing a high blood cholesterol level, treating high blood pressure, eating a healthy diet, keeping your weight in check and doing some regular physical activity can help.

Regards

Helena

But the question was "Why do arteries develop atheromas?" Not the implications of that!

This is fine Costas. I want to see the opinion of soecialists and non specialist.  A technical answer is more than welcome, but as I have shared tthe thread with a lot of non specialists it is important to know what is in their mind when they see the name atheroma. Technical answers are also more than welcome and if the ones who have given a contribution want to enrich their answers being answering exactly what was asked, it would be great.  Costas is right reminding us to be back to the core of the question.

The actions, suggested in my previous contribution to reduce the risk of forming atheroma, give an indirect answer to the question (Why do arteries develop atheromas?). The conditions of (hypercholesterolemia & hypertriglyceridemia) are often congenital but they are aggravated by a diet too rich in fats &by excessive consumption of alcohol. Cholesterol is the combination of several constituents, including HDL cholesterol, also called “the good cholesterol,” and LDL, also called “the bad cholesterol.” A lack of physical activity impairs the body's ability to “burn off” all the food eaten, and these foods accumulate, particularly in the artery walls, in the form of fats. Cigarettes are among  the main factors facilitating the development of atheromatous plaque & alcohol promotes its development. Consumption of too much sugar drops the insulin level which results in an accumulation of sugar in the blood. Consumption of more salt than needed results in an increase in hypertension. Hypertension increases the pressure on artery walls, thus promoting the deterioration of these arterial walls & the formation of atheromatous plaque.

I do wonder if "arteries suffer from atheroma", or if arteries are attacked by the growth of atheroma plaques" that destroy their inner wall and gradually disturb proper blood flow...

I 'll think it over and come back to give you a more proper answer. (I promise!)

The atherosclerotic process begins starting from endothelial cells, and then from the innermost layer of the arterial vessel.

Consider the endothelium as a simple vessel lining is very simplistic, so that today the endothelium is considered a real organ, capable of processing many active substances able to modulate the activity, not only of the various structures of the vessel wall , but also of blood cells and proteins of the coagulation system that come into contact with the surface of the endothelium. These active substances are released in the nearby (paracrine secretion), exerting its effects on the vessel wall, and partly released into the bloodstream (endocrine secretion), to carry out their action at a distance (eg. Nitric oxide and endothelin) ; others remain adherent to the surface of endothelial cells expounding its action by direct contact, as with adhesion molecules to leukocytes or for those that affect clotting.

In summary, the endothelium is the fulcrum metabolic vascular wall, to the point that regulate cell proliferation, inflammatory phenomena and thrombotic processes. For this, the endothelium plays a critical role in regulating the entry, exit and lipoprotein metabolism and other agents that can participate in the formation of atherosclerotic lesions.

The process of formation and growth of atheroma, which as we have seen develop over years or even decades, is composed of several stages: adhesion, infiltration and storage of lipoprotein particles, the inflammatory process triggered, the formation of a lesion or more less, it consists of a core lipid (lipid core) surrounded by a fibrous cap connective (fibrous cap).

To my knowledge, the buildup of plaque in arteries is not due to cholesterol, as many experts claim.

It is more likely caused by the by-products of frying, as fatty acids are fairly easily converted into radicals under heating, and those radicals recombine to make masses of unusable "gunk" that likely clog your arteries as they are being transported to the liver for breakdown. I have also heard that plaque has a high makeup of phospholipids, which makes sense as the polar portion could serve as an anchor the the membrane.

Cutting down on the cholesterol probably wouldn't hurt, though, as I can imagine it also getting stuck in the plaque if the concentration is high enough.

Dietary fiber (both soluble and insoluble) is one of the best remedies for clogged arteries.

Excuse me for so rudely trying to answer this question without doing a literature search first (and thanks to our good friend Vilemar for his admirable grasp on the concept of constructive criticism).

The answer above is not correct, but please allow me to remedy it by including results of a brief literature search as well as some new conclusions:

1. Plaques contain various oxidized lipids (including cholesterol), proteins and calcium deposits that have hardened over time with them (maybe that is where the phospholipid hypothesis came from). The bulk of most plaque is apparently protein (fibrous tissue).

2. If dietary fiber, itself, does not play roles in plaque control then the plethora of nutrients in fruits and vegetables (notably the antioxidants and probably magnesium) are most likely to be responsible for this.

3. Part of the formation of plaque is due to macrophages attempting (but failing) to rid it from the bloodstream. They only end up making it worse. This could probably be remedied by fasting, as one of the natural fasting responses is to break down unnecessary immune proteins for use in more practical roles and to use stored fatty acids for energy.

4. Increasing the concentration of HDLs in the blood stream can also combat plaque buildup because they are small and dense. These qualities allow them to dislodge plaque that they may come into contact with in the blood stream. I had previously thought that insoluble fiber also contributed to this role but I think I was just confusing the GI tract and the bloodstream. Higher-quality, non-oxidized fats increase HDL levels.

That's probably all I can contribute...

Brenda  

Every time I post a thread I start waiting for your contribution. I am happy to konw you have an intelligent husband with a good sense of humor.