Catamenial epilepsy is a type of refractory epilepsy. Much work has been done to understand the mechanism of occurrence and pathophysiology of catamenial epilepsy. Recently, extrasynaptic molecular mechanism for the targeted therapy has been suggested ; and increased seizure protection has been explained by increased antiseizure efficiency of neurosteroids models where neurosteroid sensitivity has been linked to δ GABA-A receptors.
It is generally noticed that the traditional antiepileptic drugs (AEDs) have less efficacy for catamenial seizures.
What other possible mechanisms you think might be involved in causing and exacerbating the catamenial seizures?
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