.Lipoprotein(a) - Lp(a) :
Desirable: < 14 mg/dL (< 35 nmol/l)
Borderline risk: 14 - 30 mg/dL (35 - 75 nmol/l)
High risk: 31 - 50 mg/dL (75 - 125 nmol/l)
Very high risk: > 50 mg/dL (> 125 nmol/l)
( FROM WIKIPEDIA)
Sorry, I did a mistake. 115.8 mg/dL is a normal cholesterol plasma level. The girl doesn't need a lipid-lowering drug. In this case , the statins could affect the girl health
In our hospital in China, the normal range of lipoprotein (a) is below 300 mg/L. 115.8 mg/dL is NOT a normal cholesterol plasma level. There are so many medical doctor not very familiar with lipoprotein(a).
High Lp(a) in blood is a risk factor for coronary heart disease (CHD), cerebrovascular disease (CVD), atherosclerosis, thrombosis, and stroke.
Lipoprotein(a) - Lp(a) :
Desirable: < 14 mg/dL (< 35 nmol/l)
Borderline risk: 14 - 30 mg/dL (35 - 75 nmol/l)
High risk: 31 - 50 mg/dL (75 - 125 nmol/l)
Very high risk: > 50 mg/dL (> 125 nmol/l)
( FROM WIKIPEDIA)
Sorry, when I read in the first time, I think you are asking about total cholesterol and not only a specific lipoprotein. I don't know about lipoprotein A, but I will searching and I will try help.
Thank you, Roman.
It is common that many adult patients of Nephrotic syndrome, Renal failure, Pulmonary embolism, Cerebral infarction are frequently associated with Lp(a) higher than 300mg/L. I think it is necessary to down regulate Lp(a) to prevent disease. The girl is not in good healthy.
Hi Hao Jiang, would be useful to know the rest of the lipid profile of your patients and their parents. Unfortunately there is no intervention that is really useful to lower the levels of Lp (a). However, in many patients the elevation of Lp (a) is the only recognizable risk factor after AMI. I have many patients with elevated Lp (a) similar to your patient, and if there is a significant family history of coronary disease attempted to minimize other FR, in addition to antiplatelet therapy.
Another issue is that there is an inflammatory process (some, not all, can cause elevations of Lp (a)). However, if the elevation is persistent over time, my tendency is to regard it as genetically determined and act as I said.
Thank you, Juan. Some patients with LP (a) was significantly higher than the normal range, while their TC, TG, HDL, and LDL were in the normal range. Lp(a) can be oxidized and become oxidized lipid. Many people think that it is genetically determined, but we have question. When Lp (a) is start rising? Is Plasma Lp (a) in high level when a child is born? What cause Lp(a) be elevated? Some genetically determined hormone ?