attached you can find the only international consensus on atrial cardiomyopathy.

yours, Thomas

The working group proposes the following working definition of atrial cardiomyopathy: ‘Any complex of structural, architectural, contractile or electrophysiological changes affecting the atria with the potential to produce clinically-relevant manifestations’

Diseases like a hypertension, heart failure, valvular, genetic,diabetes,

myocarditis ... or conditions (like alone AF, smoking, isolated atrial amyloidosis,ageing, endocrine abnormalities...) are known to induce or contribute to an atrial cardiomyopathy

EHRAS classification of atrial cardiomyopathy ( class/ histological characterisation)

I  Morphological or molecular changes affecting ‘primarily’

the cardiomyocytes in terms of cell hypertrophy and

myocytolysis; no significant pathological tissue fibrosis

or other interstitial changes

II Predominantly fibrotic changes; cardiomyocytes show

normal appearance

III  Combination of cardiomyocyte changes (e.g. cell hypertrophy,

myocytolysis) and fibrotic changes Alteration of

interstitial matrix without prominent

IV Collagen fibre accumulation

Iva Accumulation of amyloid

IVf Fatty infiltration

IVi Inflammatory cells

IVo Other interstitial alterations

Read more: A. Goette et al. / Journal of Arrhythmia 32 (2016) 247–278

Article EHRA/HRS/APHRS/SOLAECE expert consensus on Atrial cardiomyop...

 Thanks both of you.... for nice elaborate answar. I think CMR is very important to diagnose these cardiomyopathies... Can these cardiomyopathy produce embolism without atrial fibrillation. 

http://circgenetics.ahajournals.org/content/6/1/5

http://www.onlinejacc.org/content/65/20/2239