Is there any evidence? Is it common to see Coagulase-negative Staphylococci urinary tract infection or bacteriuria and calcium phosphate stone (apatite) instead of triple phosphate or struvite?
Thank you for your input. Is it possible that Coagulase-negative Staphylococci increases urine pH and causes apatite stones since it is on of Urea-splitting bacteria.
The association between nephrolithiasis and UTIs can be either (i) kidney stone formation developed following UTIs (the so-called ‘infection-induced stones’) or (ii) nephrolithiasis with subsequent UTIs as its complications (the socalled ‘stones with subsequent infections’). Whether they are infection-induced stones or stones with subsequent infections, the stones themselves are the important source of secondary infection. It has been thought that types of bacteria in the stone nidus implicate the causative microorganisms involved in stone formation and the pathogenesis of
‘infection-induced stones’, whereas those found in the stone periphery implicate microorganisms entrapped into the stones during secondary UTIs (stones with subsequent infections). The latter should have microorganisms found only in the periphery of stone matrices, not in the stone nidus.