Basically, the idea is to develop a molecule that could be put into the lungs as a shield so that when the virus sheds (or originally infiltrates), two of the SARS-CoV-2 attach to this one molecule and "inject each other" with their poisons. Would that be enough to slow it's progress until the immune system can overcome it? It seems like as long as these don't "settle into" the bilayer lipid membranes of the aveoli, and somehow disrupt human cells at work, it might work... What other problems exist with this concept? Thanks for any help or ideas you can contribute. - Rob
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