In advanced cases of diabetic neuropathy that have concomitant underpinning biomechanical pathology and active lifestyles, there is a pathognomonic joint destruction and collapse described by Charcot decades ago,
Root cause is neuropathy causing tendon imbalance causing excessive forces combined with lack of protective sensation. Commonest is tight Achilles causing arch collapse and breakdown of midfoot
Laborde, JM et al, Preliminary results of primary gastrocnemius-solos recession for midfoot Charcot Arthropathy, Foot& Ankle Specialist, 2015; 2(2)_:140-4
I am retired so not working with Charcot patients or any patients for that matter. However, my clinical experience and interpretation of the emerging literature is that subtle equinus is the primary mechanical etiology of Charcot arthropathy of the midfoot. Equinus is the common pathway to this pathology. Please see my 2016 article, https://www.researchgate.net/publication/305660224_The_Split_Second_Effect_The_Mechanism_of_How_Equinus_Can_Damage_the_Human_Foot_and_Ankle, especially image 4 with the accompanying text. Thanks for a great question.
Equinus is actually the primary biomechanical compensation for the inherited foot type pathology that is primary (as in functional foot typing - huge bias).
Peroneus longus and flexor hallucis longus adaptations and reduced 1st ray SERM-PERM Intervals prevent or reduce the equinus as primary care.
In advanced cases equinus must be dealt with but in pre-clinical Charcot, foot centering technology prevents the eventual Charcot bu denying the need for Equinus compensation.