I have increased expression of STAT3 in multiple sclerosis (MS) patients as compared to controls. Can Extavia be used as an inhbitor against STAT3 in MS patients? Will it be beneficial to stop the function or increased activity of STAT3? Also, since Extavia is also a protein molecule itself, then in this case protein-protein docking might be possib;e between Extavia and STAT3. How is this biologically relevant?

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