The key point is how N-succinimide is metabolized in our body. Will it cause major malfunction in the body? Does anyone know the metabolize pathway?
The main metabolite of N-succinimide is obtained through the nucleophilic opening of the imide ring. Normaly it forms succinamic acid, which is a precursor of the amino acid asparagine (with a amine on carbon 2 - (S)-2-amino-3-carbamoylpropanoic acid.
As is soluble, is excreted by kidneys .
Our, the amide group can be oxidated to carboxilic acid, forming succinic acid (a). Hidroxylation and oxidation at C2 forms oxaloacetic (b) acid which is also a precursor of arginine or aspartic acid.
I believe naked succinimide (without alkyl at N) has also substantial renal clearance as water soluble small molecule with low logP. As Ricardo wrote, presumed class 1 metabolites are quite biocompatible and possess renal clearance as well. The main question is the dose we are talking about. Traces? Grams?
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