The literature shows that myocardial antioxidant capacity, cardiac Heat Shock Porteins, Nitric Oxide, ATP-dependent potassium channel function, and opioid system activation are related to this, but also turns it inconclusive in some original articles or systematic reviews.
There are studies pointed out activation of AMP activated protein kinase may play a role.
Victor,
I would add TFPI, Tissue Factor Pathway Inhibitor. This can be released from the endothelium during and after exercise. As the name tells, this TFPI inhibits not only tissue factor but also inhibits Coagulation Factor Xa and Factor VIIa, as a result there is inhibition of the coagulation process. In short TFPI serves as an endogenous anticoagulant and thereby helps in cardioprotection.
Thanks Dr Ru-Jeng & Dr Omer,
I really conducted a search on web and found some papers revealing this points you send me. I could see it more clearly in this paper below:
Powers, S. K., Smuder, A. J., Kavazis, A. N., & Quindry, J. C. (2014). Mechanisms of exercise-induced cardioprotection. Physiology, 29(1), 27-38.
Thank you very much.
Best regards
HIF might be involved as well, see Giusti, BMC Genomics. 2009 Jul 30;10:342
you can see the page o f 174 exercise physiology book from scott k . powers and edward T howley they explain about that
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