Our group is investigating differences in the respiratory burst based on what type of biomaterial/surface the cells are attaching to. I am trying to understand what differences cause the difference in respiratory burst. What is/are the transduction molecule(s) when using PMA? Could it be that there is less, or different types of integrin/cytoskeletal attachment onto each surface? Is it the amount, or the type of attachment that affects how strong the respiratory burst is? Any suggestions of keywords or references would be great! I am still in the initial phase of the literature search.
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