Hi, I'm working on plant induced defence using methyl jasmonate. I noticed some literatures used jasmonate acid (JA) and some used methyl jasmonate (MJ). I know that MJ is the methyl ester (a derivative) of JA. But why more studies used MJ instead of JA? What is the advantage of MJ over JA when to trigger plant inducded defence? Is it that the MJ more like an "end product" than the "raw material" JA so the plant can respond faster? Or that the MJ has a better performance as a methyl ester than the JA in practice (e.g. manipuility, solubility or toxitility)?