As far as I understand, without a working version of ACE2, SARS-CoV-2 is essentially locked out of a person's immune system. Are there already some tests available on genetic forms of ACE2 that enable high inborn resistance to SARS-CoV-2 infection (similarly to CCR5-delta 32 and HIV)?
This is a very relevant question madam. We need to work on the molecular studies of this receptor to have an idea about the mutations occurring in this enzyme.
That's a very relevant question, madam. I really think that the complications due to covid 19 are partly dependent on the genetics of the host. This obviously explains that infected people don't suffer the infection in the same way, hence the interest of integrating human genetics into our thinking to better understand and predict the complications of this disease.
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