I have found two teachings from different sources. One they said about blood volume increase and therefore the patient has oedema and hypertension. But, another source says when there is high blood volume, low osmolality, it decreases the aldosterone, which promotes natriuresis and water excretion which in results euvolemic hyponatremia and oedema and hypertension does not occur? Which is true?
One of the criteria of SIADH is that the patient is Euvolemic i.e. has no fluid overload (edema, ascites, or pleural effusion).
As the symptomatic treatment involves giving oral or IV salt (sodium chloride) along with fluid restriction, BP may rise in the range of hypertension. Definitive treatment of SIADH is the treatment of the underlying cause.
For Diagnostic Criteria of Syndrome of inappropriate ADH (SIADH), glean through this link:
Article Hyponatremia: A practical approach
Thank you, no edema in SIADH in contrast to hypoproteinemic states, hyponatremia, and high urinary sodium are characteristic, fluid overload is there. However, no edema or high blood pressure.
Although SIADH is euvolemic hyponatremia, in reality patients are in the upper zone of euvolemia evident but continuously elevated urine sodium but this level of subclinical volume expansion should not be enough to give edema unless a different mechanism exist (primary salt retention) or there is chronic venous insufficiency which would precipitate lower limb edema.
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