Several papers talk about states (stressors) and pathological damage altering microglial behaviour and morphology and, as such, Iba-1 expression. However I'm struggling to find in the literature information that specifically outlines particular transmitters etc directly modulating the expression of Iba-1.

The closest I have found is ATP concentrations altering MRF-1 expression via the P2x7 receptor.

Ideas or leads? Thanks!

More Kristie L Smith's questions See All
Similar questions and discussions