hi, I hope this is useful attachment.

Thanks Masoomeh Kherad,

That's a good paper.

Best Regards

Dear Dr. Victor 

i hope the best for you , you can find the answer of your question in this researches :

* The role of nesfatin-1 in the regulation of food intake and body weight: recent developments and future endeavors

* Nesfatin‐1: a novel inhibitory regulator of food intake and body weight

* Multi-functional peptide hormone NUCB2/nesfatin-1

* Nesfatin–1: Role as Possible New Anti Obesity Treatment

* Nesfatin-1 Stimulates Fatty-Acid Oxidation by Activating AMP-Activated Protein Kinase in STZ-Induced Type 2 Diabetic Mice

* Plasma Nesfatin-1 Is Not Affected by Long-Term Food Restriction and Does Not Predict Rematuration among Iteroparous Female Rainbow Trout (Oncorhynchus mykiss)

* Nesfatin‐1 increases energy expenditure and reduces food intake in rats

* Acylated and desacylated ghrelin, preptin, leptin, and nesfatin-1 peptide changes related to the body mass index

 * Association of nesfatin-1 level with body composition, dietary intake and resting metabolic rate in obese and morbid obese subjects

best regards

moustafa

nesfatin-1 is appetite suppresent acting at the satiaty center in the hypothalamus and also mobilise fat at adipocytes. Thus it could be used for weight loss in the obese individuals.More work needs to be done o investigate if it has any side effects.

Es posible que te ayude este articulo en pubmed!

I am searching for studies which describe and examine the interaction of hormones which are released during or after exercise, that can promote apetite and obesity just in persons who want to loose weight with physical activity.

Saludos,

Benita

http://www.ncbi.nlm.nih.gov/pubmed/20625762

Eur J Appl Physiol. 2010 Nov;110(4):863-8. doi: 10.1007/s00421-010-1531-6. Epub 2010 Jul 13.

Plasma nesfatin-1 and glucoregulatory hormone responses to two different anaerobic exercise sessions.

Ghanbari-Niaki A1, Kraemer RR, Soltani R.

Author information

Abstract

Nesfatin-1 is a recently discovered anorectic protein derived from posttranslational processing of the nucleobindin 2 (NUCB2) gene. It is expressed in adipose tissue and is also found in plasma. Nesfatin-1 expression is significantly affected by nutritional status and its actions may be involved in the inhibition of the orexigenic effect of ghrelin. Although the effects of physical exercise on several anorectic and orexigenic hormones have been reported, no studies have investigated its effects upon circulating concentrations of nesfatin-1. We investigated the effects of acute strenuous interval exercise and circuit exercise on nesfatin and other hormones affected by metabolic stress. Fourteen provincial and national level young male-kickboxing volunteers participated [age 20.71 ± 2.6 years, height 176.6 ± 2.8 cm, body weight 67.2 ± 3.3 kg, and body mass index (BMI) 21.56 ± 1.42 kg/m(2)]. After an overnight fast, responses to a running-based anaerobic sprint test (RAST; 7 sets of 6 × 35 m every 10 s with 1 min rest in between sets) and a non-combat kickboxing session (NCKB; 7 sets of 6 techniques, 20 s per technique with 1 min rest in between sets) were determined. Venous blood samples were collected before, immediately after, and 45 as well as 95 min following the exercises. Plasma GH, insulin, glucose and lactate concentrations significantly increased immediately following the RAST and NCKB protocols, however, plasma nesfatin-1 concentrations were not significantly altered. Higher plasma cortisol and glucose concentrations occurred in response to the RAST compared with the NCKB protocols. Although the exercise protocols elicited metabolic stress that significantly altered circulating glucoregulatory hormones, plasma glucose and lactate, there was no significant change in plasma nesfatin-1. A lack of nesfatin-1 response to the exercise protocols may be partially due to the fasting