Especially, I would like to know changes of valvular regurgitation with asystole state. I am trying to study degree (LVEDV or LVEDP) of LV distension after acute HF.
In cases of acute HF the degree of regurgitation is underestimated, becouse of rediced CO. On the other hand it depends on the ethyology of the valve lesions and on the mechenism of HF (AMI, PE, CMP and etc)
Changes in LV dynamics and mitral regurgitation during , and post, acute heart failure are complex. They can be dependent on aetiology (ischaemic, MI, cardiomyopathy, infiltrative diseases etc.), and if there is any chronic component prior to the acute event. Generally speaking the LVESV and LVEDP will increase and the degree of mitral regurgitation will also be increased (especially if LVEDV and mitral annular size are increased) The consequent increase in LA pressure leads to increasing pulmonary venous pressures which is the driving force for pulmonary oedema.
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