Did you and Meilani Salsabila Daulay get the same homework assignment? I see you are both at the same institution, asking the same question at the same time. Maybe this is something you should find out for yourselves.

In the instructions of those drugs you can see the final comment that the precise mechanism of the action is not known. The BP lowering effect as the result of the plasma circulation volume because of the sodium and water exertion is the short-term mechanism of the action. How long the human can live with low the circulation plasma volume? The organism will restore the it shortly. These drugs decrease the sensitivity of the arterial wall to the vasopressor substation like catecholamines, angiotensin, vasopressin etc. Also, there is considering calcium entering blockade that finally results in vasodilators (Acts like calcium channel blockers). Anyway, so called "thiazide and thiazides type" diuretics are not diuretics "per se", they are the vasodilators and the best effective antihypertensive drugs with the excellent impact on the hard end-points according to the results of the majority of the clinical studies in this field.

thiazide works by minimizing sodium reabsorption in PCT. This action increases renin release, decrease cardiac output,

prevents vasoconstriction and regulate blood pressure.