How come that the virus, initially zoonotic, entered in human and transferred from human to human more efficiently than from animal to animal? Being a virus, for whom a human is a new host, SARS-2 is too generous in terms of mortality. Relatively tolerant immune answer in children to SARS-2, speaks about that cytokine storm may happen towards an intermediate loop in between the virus and immunity. My hypothesis is that virus uses normal human commensal flora to initiate its transfer from animal-to-animal and human to human. It also explains why antibiotics are efficient.
I wanted to explore the scheme above and currently am looking for a supervising lab in NY or vicinity, which can help.
Currently, there is no evidence to support that pharyngeal normal bacteria act as a carrier for SARS-CoV-2 virus.
A similar concept before was considered regarding using Mycoplasma laboratorium or Synthia bacteria as a carrier of SARS-CoV-2 virus.
There is no evidence so far, it is just an hypothesis, that is why I just wanted to doSARS pcr in different bacterial cultures from a COVID positive patient's nasopharyngeal swab. If there is no bacterial preference, than the distribution of SARS 2 among bacterial colonies will not have any preference.