With my pleasure, and all the best (in Attachment, please).

Dear Peter, Vladimir recommended two of the most important studies on the topic. You will find your answers on them.

regards

this link is useful

https://www.researchgate.net › ... › Brain › Brain Function

regards

Thanks, will take some time to read, as I am already behind. Anyway my question is related to a potential difference between physical hypertrophy, and gene expression. So resistance training can lead to increase gene expressions (or muscle memory; still need to read the articles) and not necesarely direct increase of myosin heavy chains and such. This can also be used in relation to concurrent training. Anyway, maybe I am far off with this, but at least I increase my knowledge with the mentioned articles! Thanks to you all!

As a muscle undergoes hypertrophy some myonuclei may be added to it. When the hypertrophied muscle undergoes atrophy it loses muscle mass but not myonuclei. Thus, when you retrain, the rate that you gain muscle mass will probably be faster than before initial hypertrophy as muscle tissue will have more myonuclei to synthesize protein (muscle memory).

Thanks.

Considere this, the question would relate to genes in those myonuclei related to HT (for instance myogenin, myo-D and such) If they are not kept in the myonuclei, but the myonuclei total in essential, it can be claimed that cardiovascular exercise leads to more myonuclei, and this can be used for hypertrophy also. So either "something is remembered" and altered in the myonuclei, or myonuclei number (so qualitative to quanitative) is needed. Now, of course, it can be claimed CVE would never get to high number of myonuclei in certain muscle cells. That would be a logical explanation. So concurrent training (CVE and RT) might be efficient for rather "mixed" muscle cells.

Anyway if you have no time to reply it is okay, I will read the articles soon anyway.

Thanks again