Brief Description:
Genital tract inflammation is often linked to infections (e.g., Chlamydia trachomatis, HPV, bacterial vaginosis) and dysregulated immune responses. However, the transition from acute inflammation to chronic pathology is not fully understood. Clarifying these mechanisms could reveal novel therapeutic targets for protecting reproductive health.
Mauricio Rangel Sánchez
Chronic genital tract inflammation results from persistent microbial triggers (Chlamydia, HPV, dysbiosis) and dysregulated immunity (excess Th17/IL-1β, impaired Treg/NK). This synergy drives fibrosis, infertility, and malignant transformation—making host–microbiome–immune interactions a key therapeutic target (Sturm-Ramirez et al., 2019; Łaniewski et al., 2020).
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