we want to plan a research study to determine the impact of latissimus dorsi extensibility on chronic mechanical low back pain, while intervene in combination with lumber segmental spinal joints mobilization.
Target segment will be T1 - L5.
need your feed back?
Dear Shakil,
I recommend these following papers, which I believe that deal with your question.
[1 ] Hides JA, Stokes IA, Saide M, Jull GA, Cooper DH (1994) Evidence of lumbar multifidus muscle wasting ipsilateral to symptoms in patients with acute/subacute low back pain. Spine 19: 165–172.
[2] Kim K, Lee SK, Kim YH (2010) The biomechanical effects of variation in the
maximum forces exerted by trunk muscles on the joint forces and moments in
the lumbar spine: a finite element analysis. Proc Inst Mech Eng H 224: 1165-
1174.
[3] McHugh MP, Connolly DA, Eston RG, Kremenic IJ, Nicholas SJ, et al. (1999)
The role of passive muscle stiffness in symptoms of exercise-induced muscle
damage. Am J Sports Med 27: 594-599.
[4] Chu J, Schwartz, I, Schwartz, S (2013) Chronic Refractory Myofascial Pain: Characteristics of Patients who Self-select Long-term Management with Electrical Twitch-Obtaining Intramuscular Stimulation. Int J Phys Med Rehabil, 1:4.
I hope this helps.
It is a key global back stabilizer muscle, and its tightness can cause changes in the mechanics of human spine, which may further cause pain and loss of function at lumber spine. It is also clinically very prone to tightness in human beings.
There is general agreement that RSI ( repudiative stress injury ) can lead to chronic muscle pain. A Swedish study has established the over activation of the muscle spindle cells as the basic cause.
The other mechanical injury to the muscle body ( and muscle spindle cells ) which also appears to result in the activation of muscle pain is where the tissue is stretched and as a result the spindle cells damaged - and caused to become over activated - tensing the muscle body continuously. It takes little imagination that aggressive stretching as treatment may damage tissue, - and result in further spindle cell over activation - especially when said tissue is already hyper activated and/or suffering edema.
For thousands of years gentle stretching ( such as Yoga ) and/or therapeutic high pressure - deep tissue massage have been shown to be of benefit. Heat applied has been used to relax tissue, enhance circulation and speed healing. In many Asian societies a topical "bruise medicine" of herbs in a base of oil or alcohol have been specifically developed to treat martial arts training injuries. Cold is never used, and lately in scientific research cold has appeared to reduce circulation and slow healing.
Chronic muscular pain may be linked to a previously unknown principle for how pain signals are transmitted in the human body.
This is shown by Umeå University researchers Tuija Athanassiadis and Karl-Gunnar Westberg, in collaboration with Canadian associates, in the scientific journal PLoS ONE.
"Muscles have sensory organs called muscle spindles. Their task is to inform the brain of changes in muscle length. Muscle spindles therefore contain a special type of large diameter nerve filaments that signal stretch of the muscle. The Umeå scientists' studies show that muscle spindles also contain fine nerve filaments with pain receptors. When a muscle is damaged as a result of overloading, these pain receptors are activated by the release of a signal substance from the neighboring stretch-sensitive nerve filaments in the muscle spindle.
It was previously believed that the pain receptors in muscles were exclusively found in the membranes that surround the muscles or in connection with the blood vessels in the muscle. With these new findings the Umeå researchers are drawing attention to a hitherto unknown and interesting mechanism. Damage to the stretch-sensitive nerve filaments of the muscle spindle may contribute to and sustain chronic pain in jaw muscles as well as in other muscles."
https://www.sciencedaily.com/releases/2010/06/100624091755.htm
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