Using monoclonal antibodies to remove amyloid plaques would definitely improve transmission of nerve signals. But the best treatment would be to fix the cause of those plaques, as well as of tau tangles. An article in the journal "Science Translational Medicine" ("Alzheimer's Disease: The Challenge of the Second Century", 2011) mentions the theory of an inherited factor in the disease. If this exists, the affliction could be traced back to uncorrected mutations in DNA. The proteins manufactured from the defective DNA would also be defective, possibly turning insoluble and not being able to fold correctly. These things would cause the proteins to become tightly packed and to form clumps.

Not only would this result in Alzheimer's but it could affect the eyes, leading to the lens becoming cloudy through the clumping of proteins causing cataracts. The defective proteins may even play a role in cystic fibrosis where a malfunctioning gene causes thick, sticky mucus to affect the respiratory system and pancreas. Besides the need for healthy DNA, a cell's Golgi apparatus must function properly. The Golgi organelles wrap and despatch, or package, proteins. Their normal operation is essential to avoid Alzheimer's, cataracts, and cystic fibrosis etc.