Encouraging cigarette smoking cessation and vaping cessation, increasing physical activities, starting to eat a breakfast in the morning are all lifestyle modification approaches active in the US at this time on social media.

Hi

Thank you for your reply. I agree with your detailed description for lifestyle medicine. I wish to take this discussion in little more depth like the science behind the lifestyle medicine(evidence based medicine).

1. Whether the healthcare professionals are aware of its importrance in health care delivery.

2. Do you agree or disagree that all the clinicians and health care professionals should use the lifestyle medicine as Adjunct therapy in their health care delivery system.

Can you please share your experience and views.

Drinking water and food should be free from arsenic , fluoride, nitrate, heavy metals like cadmium,lead, mercury,nikel, chromium etc.

Maintaining 25-OH Vitamin D levels of between 75 and 100 ng/ml decreases the risk of breast and prostate cancer by 75% and the risk of 8 other cancers between 30 and 70%.

Maintaining optimal ratios of Progesterone:Estradiol and also Estriol: Estrone + Estradiol as measured in salivary hormone tests also substantially reduces the risk of hormone-related cancers in women.

Also, supplementing women's diet with Indole-3-Carbinol (I3C) and Di-Indolyl Methane (DIM) steers the Estradiol detoxification pathway away from the carcinogenic 4 and 16-estrogen metabolites and toward the cancer-protective 2 -estrogen metabolites. The amount of I3C and DIM in each pill is equivalent to what you would get by eating 8 pounds of broccoli and you should take one of these pills twice daily.

Supplementing the diet with 12.5 mg/day of a combination of elemental iodine and iodide reduces the risk of breast cancer, ovarian cancer, thyroid cancer and prostate cancer. This amount of iodine/iodide is equivalent to the average amount of iodine consumed by the average Japanese citizen eating a traditional coastal diet (which contains lots of seaweed) and is 80 times more than what the average American gets in their diet! This amount is safe to consume (the Japanese have been consuming these amounts for millennia); however, it may pose some risk for those suffering from autoimmune thyroid disease.

Of course, supporting methylation pathways by taking the bio-active forms of the methylating vitamins is also very helpful in not only protecting one from cancer (through protecting DNA from damage), but also supporting many other metabolic pathways. Riboflavin-5-phosphate, Pyridoxyl-5-phosphate, 5-methyltetrahydrofolate, methylcobalamin and Betaine (Tri-methyl Glycine) are the principal nutrients involved, however there are other co-factors that are also necessary to support these pathways and include Zinc and N-Acetyl Cysteine (NAC).

The role of exposing the eyes to full-spectrum sunlight during the day and reducing or eliminating blue light exposure (such as from televisions, computers and cell phone screens) in the evening in order to help insure healthy sleep patterns helps to insure adequate melatonin production, which has very powerful anti-cancer effects also.

I hope the above information is helpful. It's been very helpful to me and to my patients in clinical practice as these are all easy and relatively inexpensive to implement.

Perm J. 2018; 22: 17-025. Published online 2017 Sep 20. doi: 10.7812/TPP/17-025 PMCID: PMC5638636 PMID: 29035175 Lifestyle Medicine: A Brief Review of Its Dramatic Impact on Health and Survival

CANCER

Despite enormous research efforts and funds expended, cancer continues to be a major cause of death. Each year, 17.5 million cancers are diagnosed and 8.7 million deaths caused by cancer occur worldwide.79 In the US, 1.6 million Americans receive a diagnosis of cancer, and more than 600,000 deaths are attributable to this disease.80 In the next few years, the world population will exceed 7.5 billion, which is expected to drive these figures even higher.81 The current belief is that most cancers are the result of inherited genetic abnormalities, yet 90% of malignancies are rooted in our lifestyle and environmental exposures. Many of these exposures are modifiable; we can avoid tobacco and alcohol, decrease our exposure to ultraviolet light, increase our level of physical activity, and, perhaps most importantly, alter our diets. In the US, October is National Breast Cancer Awareness Month; September is set aside for prostate cancer awareness; and March is dedicated to colorectal cancer awareness. These awareness campaigns are laudable; however, their emphasis on early detection, treatment, and survivorship does not address the more crucial issue that many such cancers can actually be prevented by lifestyle changes. For instance, obesity is a well-recognized risk factor for the development of a large number of malignancies, as well as for cancer recurrence and mortality.82,83 In 2016, the US will have more than 14 million persons alive as survivors of cancer. In comparison, in 1971, there were 3 million cancer survivors. By 2020, there will be 20 million people in whom some form of cancer has been diagnosed who are alive and well. More than 75% of all cancer patients currently live beyond 5 years.84 As such, there is ample time for patients to implement lifestyle changes that may further contribute to their overall disease-free, long-term survival. Colorectal Cancer In 2015, there were 1.7 million cases of colorectal carcinoma with 832,000 deaths worldwide.79 More than 140,000 people in the US will receive a diagnosis of colorectal carcinoma in 2016, and more than 50,000 will die.80 Colorectal cancer is the third most commonly diagnosed non-sex-specific cancer. Less than 20% of colorectal carcinomas have a genetic basis85; therefore, most colorectal cancer cases have been linked to environmental exposures (eg, food-borne mutagens) and chronic intestinal inflammation.86 Perhaps no malignancy other than colorectal carcinoma demonstrates so dramatically the connection between inflammation and the development of neoplasms. Patients with chronic inflammatory bowel disease (ulcerative colitis and Crohn disease) are at an increased risk of breast cancer development,87 adding to the ever-growing body of evidence linking chronic inflammation to the progression of cancer. The risk of colorectal cancer developing increases with the duration and extent of inflammatory bowel disease.87 The microbiome of the gut has also been implicated in the development of sporadic colorectal carcinoma.25,88 Risk factors for the development of colorectal cancer include a sedentary lifestyle, obesity, and the dietary components that form the basis of the standard American diet (large consumption of red meats and highly processed foods and low amounts of fruit, vegetables, legumes, and fiber intake).89 Low-fiber diets, such as the standard American or “Westernized” diet that promotes inflammation, have been linked to the increased risk and development of colorectal cancer.90 In addition, patients with colorectal cancers appear to have more comorbidities at the time of diagnosis than patients with other malignancies.91 For example, patients with diabetes have a 26% increased risk of developing colon cancer and a 30% increased risk of dying because of it compared with patients without diabetes.92 Data exist that modifiable lifestyle issues (diet and activity) are increasingly associated with the risk of colorectal cancer development, perhaps more so than any other malignancy.93,94 Prostate Cancer Prostate cancer is the most commonly diagnosed malignancy in men, affecting 1.6 million worldwide and resulting in the death of nearly 370,000 in 2015.79 In the US, nearly 150,000 men received a diagnosis of prostate cancer in 2016, and close to 40,000 will die of this cancer.80 Somewhat alarming is a recent report that the incidence of metastatic prostate cancer has increased by 72% since 2004.95 Of particular concern is that the largest increase in new cases was in the age range of 55 to 69 years, ironically the same group most likely to benefit from early treatment.96 If this increase is, as some postulate, caused by a more aggressive presentation of the disease, then the importance of lifestyle changes may further increase in relevancy. As with many other malignancies, diet and obesity contribute to chronic inflammatory processes that lead to disease aggressiveness.81,97 Furthermore, obesity has been implicated in not only the development but also the progression of prostate cancer.81,97 Prostate cancer patients are often advised to make lifestyle changes. Such changes could be beneficial but need further verification. In one study,98 changes in prostate-specific antigen levels were monitored in a small group of patients. The experimental group was subjected to an intensive lifestyle intervention program, which included a vegan diet, soy protein, supplemental vitamins (E and C), selenium, exercise (30 minutes of walking 6 d/wk), and a support group/stress management program for 1 hour per week. Prostate-specific antigen levels decreased 4% in the experimental group and increased 6% in the control group.99 In addition, blood taken from both groups demonstrated that the serum of the experimental group inhibited growth of prostate cancer cells nearly 8-fold more intensively than the serum of the control group. Furthermore, these comprehensive nutrition and lifestyle changes have been shown to downregulate prostate gene expression in men after a diagnosis of early-stage prostate cancer.99 The incidence and mortality of prostate cancer appear to be associated with a Western lifestyle, and a strong corollary relationship has been noted with the intake of animal foods.97,100 Men in developing countries who drift toward a Western-oriented lifestyle experience an increased incidence of prostate cancer. Furthermore, migration studies have demonstrated that those populations that live in low-cancer-risk geographic areas assume Western rates of cancer within 1 to 2 decades of immigration to the West. This observation is not limited to prostate cancer but involves the development of other malignancies as well.81 Breast Cancer The most common malignancy among women in 2016 was breast cancer, affecting 2.4 million women worldwide and taking the lives of more than 520,000.79 In 2017, nearly 250,000 women will receive a diagnosis of breast cancer and more than 40,000 will die in the US.80 Breast cancer is the most feared disease by many women, yet heart disease is the leading cause of death in women in the US.3 Interestingly, less than 50% of women are aware that heart disease is the leading cause of death.3 As with previously addressed malignancies, breast cancer can be attributed in part to a lifestyle fueled by a poor diet that often results in obesity.101 Sinicrope and Dannenberg101 addressed this topic in a recent publication. Obesity leads to insulin resistance, resulting in elevated blood levels of insulin and insulin-like growth factor (IGF) and a decrease in sex hormone-binding globulin. Consequently, the availability of estradiol increases, which may fuel the development and aggressiveness of breast cancers. Fatty tissues (adipocytes) have been demonstrated to be an independent endocrinogenic organ capable of manufacturing and storing estrogenic compounds that contribute to multiple malignancies. Importantly, fatty tissues also store a dozen or more inflammatory proteins that promote carcinogenesis.101 In a recent study, low-dose aspirin was shown to reduce inflammation, resulting in a lower incidence of breast cancer.102 Not only is obesity a risk factor for breast cancer development, it is an independent prognostic factor for distant metastases and an increased risk of death.103 Comorbidities also have a role in oncogenesis. Patients with diabetes have a 23% increased risk of breast cancer developing and a 38% increased risk of dying of the disease compared with patients without diabetes.92 A substantial proportion of breast cancer patients are both obese and sedentary, emphasizing the need for lifestyle interventions that may improve prognosis and ultimate outcomes.3 Understanding of breast cancer and the influence of dietary consumption patterns has recently advanced. Dietary recommendations, including an increase in fiber intake, have been made to aid in the prevention of this cancer.104,105 Recently the human “estrobolome” has been discovered as the set of gut bacterial genes that metabolize estrogen.106 Dysbiosis of the gastrointestinal tract is involved in the re-cycling of estrogen through the enterohepatic circulation, increasing its potency, which may further fuel the development of breast cancer.107,108 Estrogen levels are further decreased by the increased consumption of fiber intake because fiber inhibits the absorption of estrogen in the gastrointestinal tract.104,109 The American Institute for Cancer Research reported in 2014, from worldwide data, that diet, physical activity, and weight control are major contributors to long-term survival after a diagnosis of breast cancer.110 Furthermore, a 2011 meta-analysis of postdiagnosis exercise in patients with breast cancer involving more than 12,000 patients demonstrated a 34% decrease in risk of death caused by breast cancer, a 24% decrease in recurrence, and a 41% decrease in the risk of all-cause mortality.111 This conclusion is the result of the review of 67 published articles addressing lifestyle changes as they relate to the reduction of breast cancer recurrence.112 Additional studies have documented that physical activity not only increases survival and decreases recurrence but also improves overall quality of life in patients with breast cancer and in patients with colon cancer.113–115 Another study followed nearly 1500 women diagnosed with early-stage breast cancer. These patients were followed for up to 9 years and demonstrated a 50% decreased death rate in those who adhered to a high fruit and vegetable intake (5 servings/d) and a regular physical activity regimen (30 minutes, 5 times weekly) compared with those who did not.116 Although the exact mechanism of the action of exercise and its impact on cancer recurrence remains elusive, some have pointed to the possibility that exercise may affect the inflammatory response of the body, resulting in a decreased rate of recurrence.117 Regular exercise may also have a protective role in the initial development of breast cancer.118

Hi,

I read a lot about this issue and I can summarize the life style recommendations as follow:

1- Avoid stress

2-stop eating suger and white wheat . Replace them with natural honey and whole grains.

3-Exercise regularly4- Consume curvumin daily

5-Avoid eating red meat and replace it with plant protiens and fish

6- Avoid dairy products especially chees

Food and nutrition followed by medicinal antioxidant properties may be helpful for prevention of cancer in living beings.

hi I'm GP at Monash medical clinic( Caulfield campus)

I'm doing my Lifestyle medicine board exam in Auckland this week ( Australasian Society of lifestyle medicine )

There is plenty Evidence based facts to support need for reinforcing lifestyle education/change in almost every GP consultation.There is also big need to educate medical students and all medical professionals about the importance of this easily neglected aspect of therapy .

Thanks for reply and Good to hear from you Doctor.

To prevent cancer by

From childhood to follow good food habits

Drink more water

Daily to eat two or three mint leaf life long u cannot get cancer

Thanks for your replies.

Nitrate to nitramines from Nitrogen fertilizers and arsenic contamination in water and edible part of crop as food may cause cancer to living being. Generally ,arsenic in drinking water above 0.01 to 0.05 mg and nitrate above 50 miligram per litre may cause cancer to living beings according to WHO.