Diabetes mellitus type, also known as insulin-dependent diabetes, is a pattern of diabetes caused by the autoimmune destruction of insulin-producing beta cells in the pancreas. The resulting insulin deficiency leads to high levels of blood sugar and urine. The central symptoms are poly (frequent urination), thirst (increased thirst), nausea (increased hunger), and weight loss.
To this day, the cause of diabetes is still unknown. The distinction between type 1 diabetes and type 2 diabetes can be distinguished by self-examination. The peptide C assay, which measures the production of indoor insulin, can also be used.
Lack of care may be fatal and giving insulin is necessary to stay. Insulin therapy should continue all the time and usually does not interfere with daily activities when you are aware and well-being, as well as regularly taking dosages and blood glucose levels. People are usually qualified to manage diabetes independently; However, this may sometimes be a challenge.
Gestational diabetes is characterized by the loss of insulin-producing beta cells in the Langerhans cells of the pancreas leading to insulin deficiency. The main cause of this loss is an autoimmune immunity characterized by an attack of immune T cells on beta cells producing insulin. There is no way to prevent type 1 diabetes, which accounts for 10% of diabetics in North America and Europe (with different geographic distribution). Most people with the disease were either healthy or with optimal weights when symptoms began to appear. Their response to insulin is normal (no resistance), especially in the early stages. The first pattern can affect children or adults, but it is traditionally known to have children's diabetes because most of them are children.