I’ll answer this as a clinician. Unfortunately, I don’t think they are the same pathophysiological process. Superficial endo lends itself to Sampson’s theory of retrograde menstruation. DIE (and possibly ovarian endo) lends itself to coelomic metaplasia (Redwine’s theory). Some of the investigational markers might be the same, but the actual process and sensitivity to therapies I suspect will be different. You could look at F1 and F2 generations and you might find DIE starts to present if cell receptivity and response alters.