As the findings of this study also found hypertrophy of the type I fibers - do you think BFRRE could be used to substantially increase the ratio of type I to type II fibers and when accompanied with endurance training substantially increase the oxidative capacity of the muscle fibers? I.E. Larger type I fibers may increase mitochondrial content when endurance training is employed also. Also out of interest what rep range was used for the study?
Conference Paper Hypertrophic signaling is restored after a ten-day break bet...
I would expect to see no increased VO2max as this measure is mainly (about 70%) dominated by the oxygen transport system (cardiac output, blood volume and hemoglobin mass). If it would have an effect the accuracy of measuring VO2max limits the expected outcome (mainly due to biological variation) .It might be possible to see a little effect on LT at very low intensities (40-60%VO2max). I wouldn't be surprised if the effect appears already after one session of BFRRE. Interesting would be to evaluate the short/long term dose-effect response and a comparison to conventional training methods to improve LT. It would totally extend the very small body of literature which deals with ischemic preconditioning (IPC) for improving exercise performance. If you gonna look at it I recommend to measure the real LT and not their surrogate VT. Available for further discussions..
I suggest you both papers if you want to learn about what type of exercise increase more the VO2 max. In the first paper, the authors conclude that a 4 x 4 of HITT exercise during 8 weeks augment the VO2 max into a 7.3%.
Helgerud J, Hoydal K, Wang E, Karlsen T, Berg P, Bjerkaas M, Simonsen T, Helgesen C, Hjorth N, Bach R & Hoff J. (2007). Aerobic high-intensity intervals improve VO2max more than moderate training. Med Sci Sports Exerc 39, 665-671.
Hafstad AD, Boardman NT, Lund J, Hagve M, Khalid AM, Wisloff U, Larsen TS & Aasum E. (2011). High intensity interval training alters substrate utilization and reduces oxygen consumption in the heart. J Appl Physiol (1985) 111, 1235-1241
Hope this help you.
Thanks for your input on this. What I was trying to say was if you increase your proportion of type I fibres by volume (not by proportion) then with the correct aerobic training as well - I thought it may be possible for increased oxidative capacity of the slow twitch fibres. With increased size of non oxidative fibres from standard hypertrophy training this could lead to a drop in oxidative capacity (oxygen has greater distances to diffuse in larger muscle fibres to reach mitochondria). It would be interesting to see if any difference existed between two groups who did HIT with one employing BFRRE as well?
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