I believe that a distinction can be made. Plant disease resistance (R) genes have the ability to detect directly or indirectly a pathogen attack and facilitate a counter attack against the pathogen (i.e. some receptor proteins). The main class of R-genes consist of a nucleotide binding domain (NB) and a leucine rich repeat (LRR) domain(s) and are often referred to as (NB-LRR) R-genes which can be further subdivided into toll interleukin 1 receptor (TIR-NB-LRR) and coiled-coil (CC-NB-LRR). On the other hand, response genes are involved in metabolic pathways leading to a defense response after recognition of the pathogen (e.g. enzymes of metabolic pathways leading to ethylene, salicylic or jasmonic acid, antioxidant enzymes, etc.). Some proteins (gene products of course) with antifungal activity have also been described, which degrade the fungal mycelial wall and release elicitors that are frequently recognized by specific receptors and evoke a defense response in plants (eg. gluconases and chitinases)
I believe that a distinction can be made. Plant disease resistance (R) genes have the ability to detect directly or indirectly a pathogen attack and facilitate a counter attack against the pathogen (i.e. some receptor proteins). The main class of R-genes consist of a nucleotide binding domain (NB) and a leucine rich repeat (LRR) domain(s) and are often referred to as (NB-LRR) R-genes which can be further subdivided into toll interleukin 1 receptor (TIR-NB-LRR) and coiled-coil (CC-NB-LRR). On the other hand, response genes are involved in metabolic pathways leading to a defense response after recognition of the pathogen (e.g. enzymes of metabolic pathways leading to ethylene, salicylic or jasmonic acid, antioxidant enzymes, etc.). Some proteins (gene products of course) with antifungal activity have also been described, which degrade the fungal mycelial wall and release elicitors that are frequently recognized by specific receptors and evoke a defense response in plants (eg. gluconases and chitinases)