Yes, there is. Pain afferents from both upper cervical and trigeminal sensory fibers relay to second-order neurons of the trigeminal nucleus caudalis (caudal part of the spinal trigeminal nucleus). Many forms of headache such as cervicogenic headache, tension-type headache, migraine are associated with neck conditions including cervical spondylosis. Stabilizing and strengthening neck musculoskeletal structure is one method of preventing and managing headaches.
Copying here a paragraph from my paper linked below:
"...Pain is generated either peripherally from trigeminocervical neurons through abnormal activation at primary afferents (25) or centrally due to anomalous modulation at second-order neurons (26–28). Once nociceptive primary afferent trigeminocervical systems are peripherally activated, signals travel orthodromically and neuropeptides are released onto receptors on second- order neurons of the trigeminal nucleus caudalis (23,29,30)..."
Article The place of corticosteroids in migraine attack management: ...
There might be, but should be a diagnosis of exclusion. The diagnosis should be considered for upeer cervical degenerative diseases only, as the shared innervation of occiput and suboccipital dermatomal regions via C1 to C3 sensory nerve roots.Because the exact cause of H/A is mostly unclear and it is sought on a subjevtive background, attributing this to solely cervical spondylosis(which might be seen on imaging in as much as 60% of asymptomatic people after the 6th decade of life),needs to exclude all other causes.