I guess it depends on the purpose and model. I have seen that works in both strains. However I found in a review by Farkas (BRAIN RESEARCH REVIEWS 54 (2007) 162 – 180) that “the CA1 hippocampal subfield of Fischer 344 rats appeared to be more vulnerable to bilateral occlusion of the common carotid arteries combined with hypotension than those of Sprague–Dawley and Wistar rats”

I hope you find this report useful

L.

According to my many years of experience, there are large differences in the response of the cerebral cortex of rats to bilateral common carotid arteries occlusion. We had  monitored  the mitochondrial NADH redox state in Wistar rats up to 2 weeks after the occlusion and found that the responses to brain activation after the ischemic event are time dependent. The data suggested that the supply of oxygen to the cortex is improving in time after the occlusion. For more details please see my paper in Brain Res. 140: 217-230, 1978. Probably there are differences in the anatomical structure of the Circle of Willis in various strains.

I will be glad to help later on if needed.

Prof. Avraham Mayevsky

Hello  Hulol Alruhaimi

Related to your question. I would like to refer SD because of the vulnerability of damage caused by Ischemic model is more in Wistar. Please find the link attachment.

http://www.ncbi.nlm.nih.gov/pubmed/23103797