N-3 free fatty acids are potent hypotriglyceridemic agents. Lipoprotein remnants are TG or cholesterol rich remnants. Their catabolism and other effects may be different if there is an increase of N-3 fatty acid incorporated in to lipoprotein remnants in plasma.
Here are some contributions we have made to this area using a rat model of metabolic Syndrome. This should steer you in the right direction.
Chronic dietary n-3 PUFA intervention improves dyslipidaemia and subsequent cardiovascular complications in the JCR:LA- cp rat model of the metabolic syndrome.
PMID: 21276281
Dietary supplementation of n-3 PUFA reduces weight gain and improves postprandial lipaemia and the associated inflammatory response in the obese JCR:LA-cp rat.
PMID: 19917068
Our publications are available on my profile (Spencer Proctor).
Hi Dr Kostner. Your paper is in the list of DIABETES 53 Supp1 (2004), but that supp is'nt available. The administration group published papers will sent it to me in the next 2 weeks . Thank you for your answer, I know the relationdhip between PPAR- alfa and Fatty acids metabolism, and I am sure that your paper is very useful for us.
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