Actually I'm working on the spasticity measurement, and searching literature/ prior art for the same. I would be happy, if someone can provide me the following information:
1. What are the different types of spasticity and in which part of the body is it most prevalent?
2. What are the current measurement system that you have adopted during spastic measurement / rehabilitation, apart from MAS/Tardieu and any other manual analytical scales?
3. What is the difference between spastic stretch reflex and parkinson's rigidity reflex?
It is disappointing when you start to look at what is known about PD, to find that: (1) no-one knows what (if any) symptoms are always or usually associated and which, if any, rarely or never occur together. (2) There are no defining symptoms for PD. It used to be tremor but 30% of those with PD have no tremor and I suspect it is more like 50% because many doctors (GPs are only faced with a diagnosis a few times in their careers) have no time read research papers and would hesitate to make the diagnosis or to refer a patient in the absence of a tremor. Now it is claimed that 95% of those with PD have lost their sense of smell (http://www.medscape.com/viewarticle/714329_1) and that in those who have been diagnosed, the diagnosis should be revised if they have a sense of smell (are not anosmic). (3) Two years ago 23&Me reported that 25% of PD patients have had a joint replaced. This was never followed up.
It is therefore very unlikely that other associations have been systematically investigated. A Google search produced nothing - as no doubt you found for yourself.
I'm sorry if this sounds pessimistic but things may be about to improve! (http://www.thelancet.com/series/research)
@stephen. I'm from the mechanical stream and actually I can't capture your reply. I'm afraid that you've misinterpreted the question / I've framed the question which makes you to explain about PD. My interest is only constrained to spasticity and I framed the question to know the relation between the Spasticity and PD.
From my internet search...
1. Cog Wheel and clasp knife Spasticity. Mostly categorized as Upper / Lower extremities.
2. To measure degree of spasticity, they're using MAS / Tardieu / Clonus calculations
3. Spasticity is VELOCITY DEPENDENT stretch reflex.
My query is to about to get the elaborated information of the above mentioned questions (in title) to start the project on bio-medical instrumentation for spasticity. I think the question is clear now !
Hi,
I am coming from the PD side of things too, but I can explain the difference between spasticity and parkinsonian rigor.
As opposed to rigor, spasticity is dynamic and increases with muscle elongation. Spasticity is associated with upper motor neuron lesions. Hence, the lower motor neuron pathways are intact and the muscles reflex arc works without control from higher order structures. This leads to the mentioned stretch reflex.
I recommend you this article:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3009478/
The MAS is the most commonly score to assess spasticity.
Parkinsonian rigor is an increase in muscle tone that has it's source in the basal ganglia due to a lack of dopamine that is coming from the Substantia nigra. Therefore it is not velocity dependent, but continuously measurable. I understand that you are not specializing on PD, so I keep this short and conclude that the two phenomena are arising from different sources and are not related.
Best,
Julian
Thanks Julian .
Now I'm clear about their differences (PD and Spasticity).
I would be happy, If you/anyone could clear my another query regarding this...
As you've mentioned that the spasticity is the disorder due to the UMN lesion, I would assume that the lesion is due to aging / accidents / some other causes.. But not clear about the source of lesion.. Could you pl. help me to understand about which type of lesion which you're specifying to ?
I work with post-stroke patients. I can say the following:
1. Deep palsy spasticity replaced. This is due to upper motor neuron lesion.
2. Spasticity is formed and progresses over time. And leads to a standard position of the body (e.g. Wernicke-Mann)
3. Physiotherapy. It helps relieve simpotomy
4. Regular exercise sessions help the patient feel relaxed muscle.
5. Exercise and botulin therapy makes it possible to temporarily restore performance limbs. However, the effect does not last more than half a year.
6. The primary means of restoration is the observance of the right motor mode, exercises.
- Badges
- Science topic
- Similar topics
- Correction