In short, smoking significantly contributes to the risk of myocardial infarction (heart attack) by damaging blood vessels or increasing blood clotting: smoking increases the risk of a blood clot. If a clot forms in an already narrow coronary artery due to atherosclerosis, it can completely block blood flow to part of the heart, leading to a heart attack. Or a decrease in oxygen supply. Smoking reduces the blood's ability to carry oxygen by reducing the amount of oxygen that can bind to hemoglobin. This reduces the amount of oxygen delivered to tissues, including the heart muscle, which can lead to ischemia (lack of oxygen). Or an increase in cardiac workload. Smoking causes an increase in heart rate and blood pressure. Over time, this can increase the workload on the heart, making it more vulnerable to damage and increasing the risk of myocardial infarction.
In short, chronic smoking can lead to chronic obstructive pulmonary disease (COPD) and other respiratory problems. These conditions can put stress on the heart and lead to heart failure, where the heart muscle becomes weak and unable to pump blood efficiently.