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If multi-antibiotic resistant bacterias can escape phagocytosis, what is the actual mechanism with with it escapes and what is the relation between its antibiotic resistance and phagocytotic escape?
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I am looking to activate autophagy under autophagic inhibition conditions, by supplementing cells with extracellular calcium source, which can activate calmodulin and intern activate autophagy,...
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We are looking int calcium signaling through various calcium sources, to prevent cell death.
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HeLa cells survived and even proliferated at lower doses but SH-SY5Y. Why might SH-SY5Y cells be so sensitive? Could it be due to intrinsic properties or possible mutations in these cells?"
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I am in requirement of 4T1 cell line for cancer cytotoxicity study.
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