Cortisol is the body's chief stress hormone that also controls how your body processes carbs, fats, and proteins, and helps it to reduce inflammation. Because it's the hormone responsible for protecting you, its actions increase your appetite, tell your body to stock up on more fat, and break down materials that can be used for quick forms of energy, including muscle. Not all that you'd like to happen, but when your body senses stress (even when you know it's not the kind that will physically kill you in ten seconds or less), it thinks you won't see food again for a while -- or it may need an ample supply of fuel to camp out on during a famine or use to make a mad dash. In other words, cortisol causes tissues to break down, including muscle, skin, and collagen, while at the same time assembling fat.

LIM domain only protein 3 (LMO3) is more abundant in visceral adipose tissue of at least obese individuals and promotes adipogenesis trough PPARy upon cortisol stimulation (Lindroos et al, Cell metab.). Thus, the increased lipolysis in subcutanous adipose tissue in combination with increased adipogenesis of the visceral depot results in a redistribution  of body fat, a phenotype observed in Cushington syndrome patients.

I can suggest you a good reference:

"Deconstructing the roles of glucocorticoids in adipose tissue

biology and the development of central obesity"

Mi-Jeong Lee1, Pornpoj Pramyothin1,2, Kalypso Karastergiou1, and Susan K. Fried1

Biochim Biophys Acta. 2014 March ; 1842(3): 473–481. doi:10.1016/j.bbadis.2013.05.029

Ciao